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Rhodogil has a volume of distribution of approximately 2.7 L/kg and is only 20% bound to plasma proteins.
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Rhodogil is a centrally acting synthetic opioid analgesic.
Intramuscular (i.m.) Rhodogil increases gastric pH during anaesthesia similar to famotidine.
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Rhodogil increases the risk of CNS and respiratory depression in these patients.
Rhodogil 100 mg was statistically superior to placebo for TOTPAR, SPID, and time of remedication,
muscle relaxers) while you are using Rhodogil ; it may add to their effects.
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1 Dosing may be increased to 100 mg every four to six hours, but the daily dosage should not exceed 400 mg 1 and should be limited to 250 to 300 mg .
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This indicates that Rhodogil slightly decreases the precision of thermoregulatory control in addition to reducing the setpoint.
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Rhodogil has been shown to inhibit reuptake of norepinephrine and serotonin in vitro,
To prevent it, sit up or stand slowly, especially in the morning. Sit or lie down at the first sign of this effect.
Rhodogil may not work as well and may require higher doses to obtain the same effect as when originally taken. This is known as TOLERANCE.
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Rhodogil is administered as a racemate and both [-] and [+] forms of both Rhodogil and M1 are detected in the circulation.
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The volume of distribution of Rhodogil was 2.6 and 2.9 liters/kg in male and female subjects, respectively,
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The mean absolute bioavailability of a 100 mg oral dose is approximately 75%. The mean peak plasma concentration of racemic Rhodogil and M1 occurs at two and three hours, respectively, after administration in healthy adults.
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Ask your pharmacist if you have questions about which medicines may cause drowsiness.Rhodogil may cause dizziness; alcohol, hot weather, exercise, or fever may increase this effect.
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Rhodogil and M1 elimination half-lives (13 hrs. for Rhodogil and 19 hrs. for M1).
Rhodogil may rarely be habit forming. Rhodogil should be avoided in patients with a history.
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If you think you may be pregnant, contact your doctor. You will need to discuss the benefits and risks of using Rhodogil while you are pregnant.
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Rhodogil increases the risk of CNS and respiratory depression in these patients.
Prepare for the withdrawal symptoms of discontinued Rhodogil use.
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The analgesic activity of Rhodogil is due to both parent drug and the M1 metabolite (see CLINICAL PHARMACOLOGY, Pharmacodynamics).
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These mechanisms may contribute independently to the overall analgesic profile of Rhodogil.
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In general, both enantiomers of Rhodogil and M1 follow a parallel time course in the body following single and multiple doses although small differences (~10%) exist in the absolute amount of each enantiomer present.
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the rates of withdrawals due to adverse events appeared to be higher in the Rhodogil groups.
tests indicates that Rhodogil does not pose a genotoxic risk to humans.
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